Fibromyalgia sufferers may experience pain as a result of hypercoagulation (thickened blood) that can lead to poor circulation due to an excess buildup of fibrin.  Fibrin is a whitish, insoluble protein that makes up a blood clot when promoted by thrombin, a key clot promoter.

In otherwise healthy individuals, one burst of thrombin should generate enough fibrin to form an actual clot in the event of an injury.  However, those with fibromyalgia and other chronic conditions may continuously generate low thrombin levels that can result in hypercoagulation and pain as circulation slows.        

Reducing fibrin with the use of systemic enzyme therapy may be an alternative to taking blood thinning and pain relieving medications for those who do suffer from fibromyalgia.   

What is the role of fibrin in fibromyalgia?

Research from the late 1990s reveals that many patients with chronic disease may have an underlying coagulation defect contributing to their symptoms.  When tissues don’t receive sufficient blood, the cells are starved for oxygen and nutrients. Pain can result, as in headache or fibromyalgia.   

In the case of fibromyalgia, fibrin (a stiff protein) forms a meshwork, or screen that slows blood flow to the cells. This interferes with cell function and cell-to-cell communications leading to the accumulation of additional forms of protein debris. Therefore, the symptoms of fibromyalgia can respond well to treatments that are aimed at clearing protein debris. [1]   

Fibromyalgia sufferers may experience pain as a result of excess fibrin when promoted by thrombin, a key clot promoter.  In otherwise healthy individuals, one burst of thrombin should generate enough fibrin to form an actual clot in the event of an injury.  However, those with fibromyalgia and other chronic conditions may continuously generate low thrombin levels that can result in increased fibrin and pain as circulation slows.    

Reducing fibrin with the use of systemic enzyme therapy may be an alternative to taking blood thinning and pain relieving medications for those who do suffer from fibromyalgia.

How Vitalzym can help

Vitalzym can help relieve a variety of fibromyalgia related symptoms by addressing many of the underlying causes.

These symptoms include:

• Pain and Stiffness
• Fatigue
• Poor Circulation
• Digestive Troubles
• Irritable Bowel / Bladder

Vitalzym can also be a safer alternative to many prescription and over the counter pain relievers that may cause undesirable side effects, which is the last thing a fibromyalgia sufferer needs.

Vitalzym contains enzymes such as protease and serrapeptase, which are well-known for their proteolytic (excess protein clearing) properties. Proteolytic enzymes break down excess fibrin in your circulatory system and in other connective tissue, such as your muscles – helping to remove the meshwork that slows blood flow to the cells.   

If the rate of protein (fibrin) accumulation exceeds the rate of breakdown, the microcirculatory system gets clogged and flow decreases. This stagnation of protein debris promotes fibrosis in the muscles and other soft tissues. Combine this with slowed blood flow and the results are widespread pain and chronic fatigue.   

Vitalzym can help fibromyalgia sufferers by:   

• Acting as an anti-inflammatory agent
• Regulating the immune system
• Increasing circulation
• Reducing fibrosis
• Removing toxins and impurities in the blood
• Reducing viral loads

These enzymes also bring nutrients and oxygen-rich blood that remove the metabolic waste produced by inflammation and excess fibrin. This alone can bring tremendous relief to fibromyalgia sufferers.  

Additionally, researchers are beginning to believe that there is a strong autoimmune component connected to fibromyalgia syndrome. Interestingly enough, systemic enzyme therapy has an excellent track record regarding the treatment of various autoimmune diseases. In addition to reducing inflammation, regulating immunity, and reducing excess fibrin, serrapeptase has a profound ability to reduce pain due to its ability to block the release of pain-inducing amines from inflamed tissues.  

References

1. Hypercoagulation, Protein Accumulation, and Fibromyalgia; Keith Berndtson, M.D.; Illinois MultiMed